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Profile Publications (12)
XB-PERS-4275

Publications By James R Groome

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Acetylcholine promotes binding of α-conotoxin MII at α3 β2 nicotinic acetylcholine receptors., Sambasivarao SV, Roberts J, Bharadwaj VS, Slingsby JG, Rohleder C, Mallory C, Groome JR, McDougal OM, Maupin CM., Chembiochem. February 10, 2014; 15 (3): 413-24.


Domain III S4 in closed-state fast inactivation: insights from a periodic paralysis mutation., Groome JR, Jurkat-Rott K, Lehmann-Horn F., Channels (Austin). January 1, 2014; 8 (5): 467-71.   


Open- and closed-state fast inactivation in sodium channels: differential effects of a site-3 anemone toxin., Groome J, Lehmann-Horn F, Holzherr B., Channels (Austin). January 1, 2011; 5 (1): 65-78.


Differential effects of paramyotonia congenita mutations F1473S and F1705I on sodium channel gating., Groome JR, Larsen MF, Coonts A., Channels (Austin). January 1, 2008; 2 (1): 39-50.


Charge immobilization of skeletal muscle Na+ channels: role of residues in the inactivation linker., Groome JR, Dice MC, Fujimoto E, Ruben PC., Biophys J. September 1, 2007; 93 (5): 1519-33.


Central charged residues in DIIIS4 regulate deactivation gating in skeletal muscle sodium channels., Groome JR, Alexander HM, Fujimoto E, Sherry M, Petty D., Cell Mol Neurobiol. February 1, 2007; 27 (1): 87-106.


K-aggravated myotonia mutations at residue G1306 differentially alter deactivation gating of human skeletal muscle sodium channels., Groome JR, Fujimoto E, Ruben PC., Cell Mol Neurobiol. November 1, 2005; 25 (7): 1075-92.


Temperature-sensitive defects in paramyotonia congenita mutants R1448C and T1313M., Dice MS, Abbruzzese JL, Wheeler JT, Groome JR, Fujimoto E, Ruben PC., Muscle Nerve. September 1, 2004; 30 (3): 277-88.


Negative charges in the DIII-DIV linker of human skeletal muscle Na+ channels regulate deactivation gating., Groome JR, Fujimoto E, Ruben PC., J Physiol. April 1, 2003; 548 (Pt 1): 85-96.


Outer and central charged residues in DIVS4 of skeletal muscle sodium channels have differing roles in deactivation., Groome J, Fujimoto E, Walter L, Ruben P., Biophys J. March 1, 2002; 82 (3): 1293-307.


The delay in recovery from fast inactivation in skeletal muscle sodium channels is deactivation., Groome JR, Fujimoto E, Ruben PC., Cell Mol Neurobiol. August 1, 2000; 20 (4): 521-7.


Differential effects of homologous S4 mutations in human skeletal muscle sodium channels on deactivation gating from open and inactivated states., Groome JR, Fujimoto E, George AL, Ruben PC., J Physiol. May 1, 1999; 516 ( Pt 3) 687-98.

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