Click here to close
Hello! We notice that you are using Internet Explorer, which is not supported by Xenbase and may cause the site to display incorrectly.
We suggest using a current version of Chrome,
FireFox, or Safari.
Mol Cell Biochem
2011 Jun 01;3521-2:117-23. doi: 10.1007/s11010-011-0746-0.
Show Gene links
Show Anatomy links
Puerarin: a novel antagonist to inward rectifier potassium channel (IK1).
Zhang H
,
Zhang L
,
Zhang Q
,
Yang X
,
Yu J
,
Shun S
,
Wu Y
,
Zeng Q
,
Wang T
.
???displayArticle.abstract???
Puerarin, isolated from the root of pueraria, had been widely used to prevent and treat arrhythmia. We show that puerarin effectively prevents and reverses aconitine-induced arrhythmias in perfused heart in vitro and in rats in vivo. To study the mechanisms of antiarrhythmic action of puerarin, we investigated the electrophysiological actions of puerarin using whole-cell clamp in isolated rodent ventricular myocytes and two electrode voltage-clamp (TEV) in I(K1)-expressing Xenopus oocytes. Puerarin had no prominent effect on action potentials of ventricular myocytes from guinea pig. However, puerarin (1.2 mM) significantly inhibited the I(K1) current in rat ventricular cells. Consistently, puerarin blocked I(K1) expressed in Xenopus oocytes in a dose-dependent manner. Puerarin competed with barium, an open-channel blocker of I(K1), to inhibit I(K1) currents. Thus, our data demonstrated that puerarin is a novel open-channel blocker of I(K1), which may underlie the antiarrhythmic action of puerarin.
Chai,
[Anti-arrhythmic action of puerarin].
1985, Pubmed
Chai,
[Anti-arrhythmic action of puerarin].
1985,
Pubmed
Doyle,
The structure of the potassium channel: molecular basis of K+ conduction and selectivity.
1998,
Pubmed
Guo,
[Effect of puerarin on L-type calcium channel in isolated rat ventricular myocytes].
2004,
Pubmed
Kubo,
Primary structure and functional expression of a mouse inward rectifier potassium channel.
1993,
Pubmed
,
Xenbase
Li,
Transgenic upregulation of IK1 in the mouse heart leads to multiple abnormalities of cardiac excitability.
2004,
Pubmed
Monaghan,
Inwardly rectifying K+ currents of alveolar type II cells isolated from fetal guinea-pig lung: regulation by G protein- and Mg2+-dependent pathways.
1997,
Pubmed
Nichols,
Inward rectification and implications for cardiac excitability.
1996,
Pubmed
Noujaim,
Up-regulation of the inward rectifier K+ current (I K1) in the mouse heart accelerates and stabilizes rotors.
2007,
Pubmed
Piao,
Cardiac IK1 underlies early action potential shortening during hypoxia in the mouse heart.
2007,
Pubmed
Qian,
Blocking effect of puerarin on calcium channel in isolated guinea pig ventricular myocytes.
1999,
Pubmed
Sun,
Activation of large-conductance calcium-activated potassium channels by puerarin: the underlying mechanism of puerarin-mediated vasodilation.
2007,
Pubmed
,
Xenbase
Vaidyanathan,
Regulation of cardiac inward rectifier potassium current (I(K1)) by synapse-associated protein-97.
2010,
Pubmed
Warren,
Blockade of the inward rectifying potassium current terminates ventricular fibrillation in the guinea pig heart.
2003,
Pubmed
Wolleben,
Influence of ATP-sensitive potassium channel modulators on ischemia-induced fibrillation in isolated rat hearts.
1989,
Pubmed
Zhang,
Puerarin blocks Na+ current in rat ventricular myocytes.
2003,
Pubmed
Zhang,
Puerarin blocks transient outward K+ current and delayed rectifier K+ current in mice hippocampal CA1 neurons.
2001,
Pubmed